A healthful diet, regular exercise, and the daily use of a wide spectrum, pharmaceutical-grade nutritional supplement program, that includes vitamins, minerals, antioxidants, and the necessary fatty acids appear to be our best bet for reducing the risk of heart disease, and death from heart disease, as well as closely each other chronic degenerative impairment of normal physiological function (cancer, diabetes, lung diseases, Alzheimer’s disease …just to name a few). Antioxidants have even been shown to slow the aging process, and publicize longevity. Today, people are living longer than ever, but they are ordinarily living longer with chronic illnesses, not inevitably living longer, HEALTHIER lives.

Too oftentimes we KNOW what we “should do,” but oftentimes don’t. We get lulled into a untrue sense of “health security” plainly because we don’t “feel” anything wrong. However, the majority of humans who die of heart sickness don’t feel any sensations or changes whatsoever…they plainly have a heart attack one day and die! Even more startling, more than half of the people who die of atherosclerotic (plaque)-related heart attacks have altogether NORMAL cholesterol levels! If that is the case, then there MUST be more going on than cholesterol that is causing coronary artery sickness and deaths from heart attacks.

Our lifestyles selections have closely everything to do with our chances of dying of a heart attack, type 2 diabetes, and to some degree a lot of cancers, as well as a lot of other chronic diseases. Fortunately, we have the chance to choose amid what will aid us in getting more salubrious and what will not. For example, stress and sleep may be considered two opposing forces affecting our health. A lack of sleep and an increase in stress will increase the danger for heart disease, cancer, diabetes, and accelerate aging.

Most persons recognise to eat right, exercise, get a great deal of sleep, reduce stress, reduce exposure to toxic substances, but most persons still do not realize the unbelievable gains that may be received through every day implementation of a full spectrum, pharmacuetical-grade vitamin, mineral, antioxidant, and omega-3 fatty acid nutritional supplement program.

The True Cause of Heart Disease and Stroke

Atherosclerosis is a disease characterized mainly by inflammation of the arterial lining caused by oxidative harm from homocysteine, a toxic amino acid complex found in everyone. Homocsyteine, in combining with other free radicals and toxins that we’re all continually exposed to (stress, pollution, unhealthful feed choices, cigarette smoke, etc.), oxidizes arteries, LDL cholesterol, and triglycerides, which in turn releases C Reactive Protein (CRP) from the liver-a marker of an inflammation. Inflammation (which results from oxidation) is the beginning stage of plaque build-up and ultimately, cardiovascular disease. Plaque, combined with the thickening of arterial smooth muscles, arterial spasms, and clotting, puts a person at a high peril of suffering heart attack or stroke.

Oxidation of metal resuls in rust. We’re not made of metal so we do not rust, but we do undergo oxidation. Cut an apple in half, and watch it turn brown when it is exposed to air ….it is being oxidized, or damaged. Bananas when purchased green at the grocery store and put on your kitchen counter, will ultimately ripen to yellow. Oxidation causes them to turn brown, and then black. Rubber bands left in the driveway in the hot summer sun, exposed to UV light, become brittle after a few weeks as the elastic corpuscles are oxidized (damaged). People who do not protect their skin from the sun and exccessively tan, cause oxidative harm to collagen and elastic molecules, and end up wrinkled more than others who protect their skin.

Oxidation occurs each single second all around our entire body, organs, and cells. Free radicals, from respective sources, including breathing and metabolism, let alone from toxic chemicals, stress, trans fat, and cigarette smoking, strip away electrons from cell membranes, hormone receptors (including insulin receptors), lipids (including cholesterol), enzymes, and DNA …which may initiate cancer.

Antioxidants, which are both devised by your cells and ingested in the form of vitamins and other supplements, “donate” electrons to neutralize free radicals, and accordingly protect our cells and cellular structures from damage. Our cells are protected by antioxidants, and they repair themselves. Healthy cells function at their optimum. Healthy cells not only function normally, healthful cells are, by definition, not cancerous.

This does NOT mean that a person who takes nutritional supplements will never create heart disease, stroke, cancer, diabetes, Alzheimer’s disease, wrinkle, or die. However, this much is true …just like we know, persons who smoke 2 to 4 packs of cigarettes per day (excessive oxidation) have an increased peril of almost all chronic impairment of normal physiological functions equated to those who don’t smoke, and likewise, those who do not take a full-spectrum, quality nutritional supplement program have a much better chance of living a longer and more salubrious life equated to those who do not adequately supplement.

Regarding the number one cause of death, heart disease: Of the two major types of cholesterol, HDL and LDL cholesterol, the more indispensable parameter is the level of HDL cholesterol. HDL, or high-density lipoprotein cholesterol, is responsible for clearing out the LDL cholesterol that sticks to arterial walls. Exercise, vitamins, minerals, and other antioxidants, specially the bioflavonoid and olive polyphenol antioxidants, increase HDL cholesterol levels and protect the LDL cholesterol from oxidative damage, and accordingly may do more to reduce your danger of heart sickness than medications.

LDL cholesterol is not inherent bad. It is necessary for life. It is indispensable for numerous cellular functions, hormone production, cellular repair, brain function, etc. LDL cholesterol only becomes “bad” when it is damaged, or oxidized by free radicals. Only the damaged, or oxidized form of LDL cholesterol sticks to the arterial walls to initiate the formation of plaque. So, rather than focusing on letting down the level of LDL cholesterol, it make more sense to focus on letting down the OXIDATION of LDL cholesterol!

Let us look at the case of cigarette smoking for a simple example demonstrating that we in truth need to reduce oxidized LDL cholesterol to prevent atherosclerosis, as opposed to indiscriminately letting down LDL cholesterol with statin drugs. Everyone knows that cigarette smoking increments the risk of some chronic diseases, such as cancer, heart disease, and stroke. Smokers with normal levels of LDL cholesterol are at an even dandier peril of fabricating heart sickness than a non-smoker who has elevated levels of LDL cholesterol. Of course the reason why a smoker with normal levels of LDL cholesterol is at dandier danger of disease is because his LDL gets excessively oxidized.

Cigarettes and antioxidants are opposites. Everything that cigarette smoking would do to injure you, and HOW it harms you, is precisely the opposite of everything antioxidants do to protect you and HOW they protect your cells. If cigarettes increase the risk of heart disease, antioxidants support reduce the peril of heart disease. If cigarettes increase the peril of cancer, antioxidants aid reduce the peril of cancer. If cigarettes increase the risk of wrinkling of the skin and aging of the body, guess what does the opposite? Yes, antioxdidants!

Certainly, it is true that if you have two people, and neither of whom are taking nutritional supplements, the one with the most eminent cholesterol level would “lose;” that is have the more outstanding risk of heart disease, heart attack, and death. The reason for this, he would have more LDL cholesterol getting oxidized and sticking to the arterial walls than the person with lower amounts of LDL cholesterol, even though the person with lower levels of LDL cholesterol is still constructing heart disease (just at a slower rate than the person with a higher level). However, studies suggest it may be possible to have a person with a higher LDL cholesterol level than another person, but because he takes a full range of pharmaceutical-grade nutritional supplements he may be at lower risk of heart sickness than the person with normal LDL cholesterol levels who does not take supplements.

Vitamins, Minerals, Antioxidants and Essential Fatty Acids: The Real Way to Maintain CardioVascular Health

The answer to letting down the life threatening risk of arteriosclerosis, heart sickness and stroke is to reduce the oxidative harm to the LDL cholesterol, triglycerides, and the arterial lining so that inflammation and resultant plaque does not form. Oxidation and inflammation are efficaciously neutralized with the use of vitamins, minerals, antioxidants, and omega-3 fatty acids; and the medical studies show that less oxidative harm and chronic inflammation in the body translates into less incidence of closely all chronic diseases. It is closely that simple. However, we will have to do more than take nutritional supplements to protect ourselves from heart disease; we ought to eat right, exercise, get adequate sleep, and reduce chronic stress as much as possible. And, it is my opinion that the answer to the preventative action of heart disease does NOT lie in statin drugs.

Not long ago, widespread interest was ignited by studies that reported associations among antioxidants and reduction in death from heart disease, stroke, and cancer. Interest continued to increase as even larger studies were published by researchers from Harvard Medical School, who saw a link amid higher vitamin E blood levels and lower numbers of cardiovascular deaths. As time goes on, the more we are learning when it comes to the immense gains of respective antioxidants, as well as minerals, in reducing the danger of developing most chronic degenerative diseases, including cancer, diabetes, and Alzheimer’s disease.

Vitamin E is a fat-soluble vitamin. It helps protect LDL cholesterol, as well as all cell membranes in all organ tissues from oxidative damage. Vitamin E rides along with the triglycerides and cholesterol protecting them from oxidative damage. Recent studies show that the natural form of vitamin E is competent to reduce the danger of heart impairment of normal physiological function and atherosclerosis beyond it is antioxidant effect. (The full range of the natural form of vitamin E includes: d alpha tocopherol, gamma tocopherol, delta tocopherol, and the tocotrienols.)

Knowing all the gains of vitamin E, may you imagine my shock in 2005 when I read in one of my medical journals a three page report when it comes to the results from the Nurses Health Study that vitamin E was “shown NOT to reduce the danger of heart disease?” This was a powerful and statistically substantial study, and the main report was genuinely when it comes to how aspirin scaled down the danger of stroke in healthful women by 24%. I read that and thought to myself, “okay, I may buy that. I see how aspirin, which thins the blood may reduce the danger of stroke, …and 24% is statistically significant.”

I likewise know that aspirin kills over 21,000 humans in the United States alone due to gastrointestinal bleeds. The shock came when I saw on the third page of the report, a little headline with regards to this study’s corollary findings. The headline read, “Vitamin E Shown Not to Reduce the Risk of Cardiovascular Heart Disease.” I couldn’t believe it. I wondered how this could be, after 30+ years of studies showing the gains of vitamin E in sustaining a healthful heart.

Since I knew better, I necessitated to read further. Sure enough, from the Nurses Health Study when it comes to the preventative action of heart impairment of normal physiological function and vitamin E, the report stated …and I’ll quote, “vitamin E showed neither any hurt or gain in all the clinical parameters examined.” My firstborn thought was, good, I’m glad there was no injure …but, how could it be that there was NO benefit? This sounded very definitive. “NO BENEFIT IN ALL THE CLINICAL PARAMETERS EXAMINED!”

Then, I read the very next sentence, “The one exception, vitamin E was shown to confer a 24% decreased danger of cardiovascular death.” As if I was not stunned sufficient already, after reading this I was actually confused. “Wait a minute? ….the former sentence said that vitamin E showed no gain in all the clinical parameters in reducing the peril of heart impairment of normal physiological function ….the one exception being a 24%, decreased risk of cardiovascular death!” Decreasing the peril of cardiovascular death is rather a benefit, wouldn’t you think?

Look at it this way, aspirin scaled down the risk of stroke by 24% and it got all the praise, in spite of it being responsible for killing 21,000 persons each year in the U.S. Vitamin E showed no hurt whatsoever, and it scaled down the danger of cardiovascular death by 24%, and someways this was of no benefit?

I figured out what was going on. Vitamin E did not show any gain in all the clinical parameters examined, …you see, I encountered that death was NOT one of the clinical parameters of the study. They just happened to detect that there was a 24% decreased danger of death. The parameters of the study were things like blood pressure, heart rate, arrhythmias, cholesterol levels and ratios, etc. Vitamin E did not modify these parameters …all it did was prevent DEATH! Since it did not show any gains according to the parameters of the study the writers concluded it didn’t do anything …except prevent versus death.

The researchers stated that they were unable to explain the scaled down risk of death from heart disease. I may explain it …it’s simple: vitamin E protects cholesterol from oxidation. Cholesterol that is not oxidized does not stick to the arterial walls, and if it doesn’t stick to the walls, there is less atherosclerosis, and less atherosclerosis means less heart attacks, and less heart attacks, translates into a scaled down danger of death. Can you imagine what the researchers would witness if they had followed the share of oxidized cholesterol levels? If they would have tested for lipid peroxidation, that is oxidized cholesterol and triglycerides, they would have seen a unfathomed reduction in this “parameter.”

This Nurses Health Study involving vitamin E was the greatest and longest study ever conducted in regards to vitamin E among women. Published in the Journal of the American Medical Association in 2005, this study involving over 40,000 women found that vitamin E scaled down the risk of death from cardiovascular impairment of normal physiological function in women underneath the age of 65 by 24%, …and even more impressive, in women over the age of 65, vitamin E was shown to reduce the danger of DEATH by an astounding 49%!

A study published in the 1996 report from the National Institutes of Health (Division on Aging), showed impressive results amid a study population of 11,178 persons using multiple antioxidant vitamins who were followed for ten years. The group of elderly persons that supplemented with vitamins E and C above the amounts found in a mutual multi-vitamin had one-half the number of deaths from heart impairment of normal physiological function of those who did not supplement. That is a 50% reduction in the number one cause of death!

Furthermore, the supplemented group demonstrated less overall deaths from NEARLY ANY CAUSE, INCLUDING CANCER, which was decreased by closely 60%. Not one study involving drugs comes close to showing such gains (and without the risks of drugs). Even more impressive, these gains were found with the use of only a few antioxidants. Can you imagine the gains possible from using a full range of quality supplements?

Antioxidants provide shelter from some diseases. B-complex vitamins, folic acid and B6, are related with a scaled down risk of heart attack. Researchers likewise believe that vitamin C provides shelter to arterial walls and reduces plaque formation by neutralizing free radicals before they may harm elastic tissues and affect cholesterol profiles. The epidemic deficiency of vitamin D is related with an increased peril of heart disease, and supplementation with at least 1000 IU of vitamin D revereses this deficiency and provides a heap of and aweinspiring health benefits. Others have studied the usefulness of a combining of antioxidants, in particular vitamins E, beta-carotene, and vitamin C in the preventative action of heart disease and found them to be beneficial. In addition, bioflavonoid antioxidants are affiliated with a reduction in some diseases, including cardiovascular disease. Add to this gains of co-enzyme Q10 and olive phenol antioxidants-and one may genuinely gain beyond taking any medications for prevention.

Beta carotene, grape seed extract and olive extract penetrate deeper inside the LDL cholesterol and arterial walls, adding more shelter from oxidation. Quercetin and alpha lipoic acid work through nitrous oxide pathways to reduce high blood pressure and prevent spasms of coronary arteries, both being major risk components for heart disease, heart attacks, and death.

Studies have shown how the B-complex vitamins, exceptionally folic acid, vitamin B6, and vitamin B12 help reduce homocysteine levels by palliating the conversion of homocysteine into the safe amino acid, cysteine. This leaves less homocysteine to oxidize and nick the arterial walls and LDL cholesterol. To further reduce homocysteine levels, one may take the amino acid betaine, likewise known as tri-methyl-glycine (TMG), as it “back-converts” homocysteine into the safe amino acid, methionine.

Vitamin C intake is related with a decreased peril of heart disease. One study found that the risk of death from cardiovascular impairment of normal physiological function was 42% lower in men and 25% lower in women who consumed more than 300 mg/day. Recent results from the Nurses’ Health Study, based on the follow-up of more than 85,000 women over a sixteen-year period, also suggest that higher vitamin C intakes may be cardioprotective. Vitamin C and grape seed extract in combining are very powerful in preventing oxidation in all areas; the cholesterol, the endothelium, and deeper in the arterial walls. Furthermore, the two antioxidants replenish vitamin E and other antioxidants to synergistically protect cholesterol and the arterial walls.

The bioflavonoid class of antioxidants such as grape seed extract, resveratrol, and quercetin, has been related with reducing the danger of heart attack and stroke by as much as 50% and perchance as high as 70%. Grape seed extract and resveratrol are two potent antioxidants that neutralize free radical oxidation of cholesterol, the arterial lining, the arterial wall, the liver, the brain, the kidneys, and the skin. Both work synergistically with other antioxidants.

Studies have demonstrated the capacity of bioflavonoid antioxidants such as quercetin, to reduce high blood pressure, a well-known danger element for heart attack and stroke. Doctors commonly turn to antihypertensive medications to lower blood pressure as the firstborn line of treatment for hypertension rather than through safer means. Diet, exercise, weight loss, and the use of vitamins and solid homogeneous inorgani substances have all been shown to reduce high blood pressure. If persons genuinely knew how powerful grape seed extract, resveratrol, alpha lipoic acid, and quercetin are in reducing LDL oxidation, increasing HDL levels, strengthening arteries and capillaries, and reducing high blood pressure each person would be taking these nutrients.

Green tea extract helps to both reduce the oxidation of LDL cholesterol and to lower LDL cholesterol. Both actions are necessary, not just the letting down of LDL cholesterol, as with prescription drugs. Green tea extract likewise reduces the oxidation of the endothelium (the thin layer of cells that line the heart, blood vessels, etc.) Green tea extract increments HDL cholesterol and it helps prevent cancer.

Olive extract has been shown to be even more effective in protecting LDL cholesterol and reducing the risk of heart disease and cancer than most other antioxidants, but it’s more effective if employed in combining with a myriad of antioxidants already mentioned.

Soy isoflavones have been shown to reduce the danger of heart disease. A meta-analysis of eight randomized, controlled clinical tryouts published in the September 1, 2004, issue of the Journal of Nutrition found that soy isoflavones were successful in letting down serum low-density lipoprotein (LDL) cholesterol. Again, letting down LDL cholesterol is secondary to the necessary protective means of protecting LDL from being oxidized; but the action of soy includes cholesterol lowering, plus inhibiting LDL oxidation, protecting versus development of atherosclerosis, and reducing peril of thrombosis. The active elements in soy responsible for these gains are the isoflavones: genistein, daidzein, and glycitein. Soy along with Co-Q10 has been shown to reduce risk of breast cancer.

Alpha-lipoic acid protects versus oxidative harm to LDL cholesterol and reduces the inflammatory cellular response within arterial walls that gives rise to more oxidative harm and plaque formation. Alpha-lipoic acid along with magnesium is capable to prevent and support reduce high blood pressure in diabetics. This happens by raising the levels of glutathione to protect bio-mechanisms that regulate arterial tone. Furthermore, alpha-lipoic acid prevents reperfusion injuries after ischemic injuries, i.e., harm done to tissue when the blood flows back into damaged vessels and leaks into the heart muscle after heart attacks.

The combining of alpha-lipoic acid (ALA), vitamin E, grape seed extract, resveratrol, olive extract, green tea extract, quercetin, and Co-Q10 synergistically work together in the preventative action of oxidation of LDL cholesterol and the arteries much better than any working alone. All these antioxidants likewise aid reduce the risk of cancer by inducing apoptosis, the “self destruction” of cancer cells.

Olive oil and olive extract bestow unbelievable health benefits. There are peculiar powerful phenol antioxidants within the fruit of olives that are related with a substantial decreased risk of heart attack and cancer. The Mediterranean diet, in which olive oil is the important source of fat, has been shown to provide the longest lifetime and lowest incidence of heart disease and cancers. Many studies have shown olive oil reduces death from heart sickness by reducing atherosclerosis, plaque formation, letting down LDL cholesterol, preventing oxidation of LDL cholesterol, raising HDL cholesterol, and preventing unnatural and potentially lethal clot formation.

Researchers found that countries in which humans consumed the most eminent amount of olive oil had the lowest incidence of heart disease and cancers. People of Greece were found to have the most eminent consumption of olive oil, in particular those on the island of Crete. On Crete, persons traditionally consume 2 to 3 oz. of olive oil per day.

On the island of Crete heart impairment of normal physiological function and cancers have been rare; 37 times (not 37 percent, but 37 times) lower incidence than that found in the United States! In essence, we may think of heart disease and cancers as rare on Crete as scurvy and rickets are in the United States.

Anyone could consume 2 to 3 oz. of extra virgin olive oil each day to obtain these same benefits, but that would add when it comes to 500 to 750 calories to one’s diet and cost an further and added $100 U.S. per month. Then, there’s the problem that not all olive oil brands are consistent in their concentrations of antioxidants. The best way to get olive phenol antioxidants is to take a standardized, pharmaceutical-grade olive extract.

Are All Vitamins the Same?

In the last decade, the dietary supplements industry has grown tremendously. The fact that nutritional supplements do not receive the same regulation as prescription or over-the-counter drugs raises questions in the minds of a good deal of consumers. That’s why NSF became involved in devising the nation’s introductory genuinely independent testing usual and product certification program strictly for dietary supplements. Consumer Labs is another independent source assessing the quality of nutritional supplements.

Due to the independent testing and guaranteed pharmaceutical Good Manufacturing Practices I am convinced of the amount and quality of ingredients I am taking. Within my multi-antioxidant, which already has a full spectrum of other antioxidants, I get 30 mg of olive extract. The 30 mg of olive extract is equivalent to consuming 2 to 3 oz. of extra virgin olive oil per day. That would mean, even though I live in the U.S., I get the same gains as those on the island of Crete who have 37 times lower danger of heart disease and cancers than in the U.S.

Fish oil supplements: necessary fatty acids, peculiarly the type that comprise concetrated levels of DHA (docosahexaenoic acid) along with EPA (eicosapentaenoic acid), reduce oxidation of LDL cholesterol and inflammation of arteries, and thereby protect the heart. Most commercially available fish oil supplements consist principally of EPA, yet scientific proof shows it is the DHA element that is more effective in preventing heart disease. Worse is the concern for mercury – which is more of a problem with eating fish. The best counsel I may give is to find a pure fish oil supplement with concentrated DHA levels.

Once again, let us compare the effectiveness of statin drugs with something as simple as fish oil or flaxseed oil supplements. Statin drugs lower LDL, but that has not translated into significantly less heart attacks and death, because letting down LDL cholesterol is not the key to preventing disease.

However, the omega-3 necessary fatty acids, the parts in fish and flaxseed oils that make them so beneficial, actually do decrease death and disease. Fish oil and flaxseed oil reduce ventricular dysrrhythmias, platelet aggregation, and triglycerides. These oils serve to decrease the number of immune cells that cohere to arterial walls, and therefore reduce plaque formation. They likewise increase nitric oxide production, which lowers blood pressure. And, they reduce oxidation of LDL cholesterol!

Omega-3 fatty acids, as found in nuts, oily fish, and flax seed support protect LDL from oxidation, and accordingly act like “antioxidant” fats. These fats do not cause damage, do not solidify, and do not stick to the walls of the arteries. They get integrated into LDL cholesterol and protect it from being oxidized. Thus fish and flaxseed oil reduce the risk of atherosclerosis. There is no comparison amidst the “benefits” of statin drugs and the actual gains seen with omega-3 fatty acids.

In a study consisting of 11,323 persons who had suffered a heart attack within the former three months, fish oil supplements (850 mg of EPA=DHA) were shown to reduce the peril of sudden death and repeat heart attacks by 45%, and provide a 20% decrease in total mortality, versus placebo. Statin drugs don’t come close to fabricating those results. Additional studies assert these powerful and significant results.

A report published in the Archives of Internal Medicine in 2005 looked at 97 double-blind controlled studies comparing the efficacy of cholesterol letting down statin drugs to fish oil. They found that cholesterol-lowering statin drugs scaled down the danger of death from heart impairment of normal physiological function by only 13%, and interesting sufficient it was NOT due to the effect of letting down cholesterol. The benefits, altho small, were derived from the fact that statin drugs have a slight to modest antioxidant effect.

Even more interesting, the salmon oil was shown to reduce the risk of death from heart disease by 23%, almost double the gain of statin drugs. Salmon oil is an omega-3 fatty acid that gets integrated into cholesterol and triglycerides and prevents the oxidation of LDL cholesterol. Since LDL cholesterol is protected from exuberant oxidation there is less plaque build up and less danger of heart disease.

Magnesium relaxes smooth muscles in the arterial walls, thence opening up arteries. It also helps incorporate calcium into the bones when intake of calcium and magnesium are the right way balanced, as in a good supplement. Magnesium helps maintain the rhythm of the heart.

Magnesium helps to reduce the risk of arrhythmias and other electrical dysfunctions following a heart attack.

Damage that leads to unnatural heartbeats is worsened by magnesium deficiencies. In addition to magnesium supplementation, researchers are finding promise in the higher rates of survival after heart attacks when high-dose antioxidants are commenced shortly after an attack. Apparently, antioxidants support prevent the mechanisms that lead to vasospasm (the spasm and closing off of little arteries) and blood clot formation. Magnesium may even support reduce the incident and severity of migraine headaches, particularly in combining with the other vitamins and minerals, in particular the grape seed extract and turmeric extract.

Fiber is necessary in the diet in order to reduce the peril of atherosclerosis. Fiber helps to control the rate at which sugar enters the bloodstream. Diets high in both soluble and insoluble fiber help reduce blood lipid levels, cholesterol, and insulin resistance. Most humans hardly consume five grams of fiber per day. However, our target will have to intermediate around 30 grams of fiber per day. High fiber and less simple and starchy foods are what constitutes a healthful “low-glycemic” diet. (For more data on a low glycemic diet, or weight loss program, please see the Weight Management section.)

The thought of taking some pills each day may be rather daunting for a great deal of people. Therefore, when I practiced medicine, and even now, my recommendations are for persons to take a full spectrum, pharmaceutical brand vitamin and solid homogeneous inorgani substance supplement that holds all the necessities that one needs in as few tablets as possible. Of course, there will be other supplements you will want to add to the necessary vitamin and solid homogeneous inorgani substance foundation, such as omega-3 fatty acids, grape seed extract, and co-enzyme Q10. However, if your necessary vitamin and solid homogeneous inorgani substance program is wide sectrum and completely balanced, then you will be getting most of the supplements discussed in this article.

To reduce the peril of chronic impairment of normal physiological functions it is critical to eat right, exercise, reduce stress, obtain adequate sleep, and supplement your diet with the rectify nutritional supplements. Most supplements do not comprise the amount or purity of ingredients listed on the bottle. Many supplements do not dissolve as they should, nor are readily abosrbable, …and if they are, they may not be in the most bio-available condition for the cells to utilize. You need to trust the company that is manufacturing the supplements that you suppose to support you maintain good health for a lifetime. You need to trust the potency, purity, and bio-availability. You must talk about all medical conditions with your physician, however, you may need guidance in regard to finding a quality nutritional supplement brand.

Get healthful and stay healthful with the principles discussed in this article. Applying these principles will mainly increase your prospects of living a long AND healthful life.

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“All truth passes through three stages.

First, it is ridiculed.

Second, it is violently opposed.

Third, it is accepted as being self-evident.”

Arthur Schopenhauer

(1788 – 1860)

What is the unfeigned cause of heart disease, and how may we veritably reduce the danger of death?

Atherosclerosis, or Coronary Artery Disease (CAD), is the leading cause of death in both men and women. In the U.S. alone, there are more than one million heart attacks each year, one third of them resulting in death. The majority of men and women presently have, or are actively developing, atherosclerosis. By age 20, most persons already have a 15-25% narrowing of their arteries due to plaque formation. By age 40, there is a 30-50% clogging of their arteries.

In the beginning of the Twentieth Century, congestive heart sickness (CHD) was for the most part a result of rheumatic fever, which was a childhood disease. However by the year 1936 there was a dramatic change in the main cause of heart disease. Cardiovascular disease caused by atherosclerosis, or plaque buildup, took initial place as the important cause of heart disease, making congestive heart failure a distant second.

During the 1950′s, the autopsies conducted on men who passed from physical life of heart sickness that revealed plaque-clogged arteries concluded that cholesterol was the cause of hardening of the arteries (atherosclerosis) and coronary artery disease. Cholesterol, not calcium, was considered the “cause” of heart disease, in spite of plaque consisting of 95% calcium and a comparatively little percentage of cholesterol. By 1956 there were 600,000 deaths each year from heart impairment of normal physiological function in the U.S. Of those 600,000, 90% were caused by atherosclerosis, or clogged arteries. In less than 25 years, the number one cause of death in the U.S. had changed dramatically …from congestive heart sickness to coronary artery disease.

Because cholesterol was dubbed the “cause” of atherosclerosis, the crusade to lower cholesterol by any means started out in earnest. Both the feed industry and the pharmaceutical industry seized upon this prospect to cash in on a cholesterol-lowering effort by creating foods and drugs that would supposedly save lives. Diets, such as the Prudent Diet, were traditionalisti to lower the amount of cholesterol intake from food. There was no doubt that both polyunsaturated oils and drugs scaled down cholesterol, but by 1966 it was likewise apparent that letting down cholesterol did not translate into a scaled down peril of death from heart disease.

As there was so much cash to be made from pharmaceutical development, the venture to construct cholesterol-lowering drugs kicked into high gear, in spite of the lack of proof showing that the letting down cholesterol scaled down the peril of untimely death from heart disease.

Heart sickness kills 725,000 Americans annually, with women accounting for 2/3 or almost 500,000 of those deaths. After thirty years of cholesterol-lowering medications’ failure to significantly lower the death rate from cardiovascular disease, in 1987 a new and more dangerous class of drugs was unleashed upon the world: the “statin” drugs. Cholesterol-lowering statin drugs are now the general of care that physicians are indoctrinated into prescribing to reduce cardiovascular disease. Are statin drugs the best way to prevent heart attacks and death?

Before 1936 the most mutual type of heart disease was congestive heart sickness (CHD). It seldom caused sudden death and could be treated with the drug digitalis. The incidence of CHD remained stable until 1987, after which the incidence of the sickness skyrocketed. Interestingly, the timing of the increased incidence of congestive heart impairment of normal physiological function coincides with the introduction of cholesterol-lowering statin drugs. Could cholesterol-lowering statin drugs have something to do with the weakening of heart muscles and the increased incidence of congestive heart failure? We will see that letting down the body’s co-enzyme Q10 levels, a side effect of statin drugs, does in truth increase the danger of muscle damage, including the muscles of the heart.

Atherosclerosis is a disease characterized principally by inflammation of the arterial lining caused by oxidative harm from homocysteine, a toxic amino acid intermediary found in everyone. Homocsyteine, in combining with other free radicals and toxins, oxidizes arteries, LDL cholesterol, and triglycerides, which in turn releases C Reactive Protein (CRP) from the liver-a marker of an inflammatory response within the arteries. Inflammation (oxidation) is the beginning of plaque buildup and ultimately, cardiovascular disease. Plaque, combined with the thickening of arterial smooth muscles, arterial spasms, and clotting, puts a person at a high danger of suffering heart attack or stroke.

For years, doctors have hyper-focused on cholesterol levels. First it was the total cholesterol; later the focus became the ratio of “good” HDL cholesterol to “bad” LDL cholesterol. In other words, how much of your cholesterol was good, and how much was bad? Of the two, the primary parameter is the level of HDL cholesterol, not LDL cholesterol. HDL, or high-density lipoprotein cholesterol, is responsible for clearing out the LDL cholesterol that sticks to arterial walls. Exercise, vitamins, minerals, and other antioxidants, particularly the bioflavonoid and olive polyphenol antioxidants, increase HDL cholesterol levels and protect the LDL cholesterol from oxidative damage, and consequently do more to reduce the risk of heart impairment of normal physiological function than any medication ever could.

There is not one thing inherent bad regarding LDL cholesterol. LDL cholesterol is critical to maintain life. LDL cholesterol only becomes “bad” when it is damaged, or oxidized by free radicals. Only the damaged, or oxidized form of LDL cholesterol sticks to the arterial walls to initiate the formation of plaque.

Let us look towards cigarette smoking for a simple example demonstrating that we in truth need to reduce oxidized LDL cholesterol to prevent atherosclerosis, as opposed to indiscriminately letting down LDL cholesterol with statin drugs. Everyone knows that cigarette smoking increments the danger of a good deal of chronic diseases, such as cancer, heart disease, and stroke. Smokers with normal levels of LDL cholesterol are at an even more outstanding risk of constructing heart impairment of normal physiological function than a non-smoker who has elevated levels of LDL cholesterol. Of course the reason why a smoker with normal levels of LDL cholesterol is at dandier danger of sickness is because his LDL gets excessively oxidized.

Cigarette smoke releases so a great deal of toxins and free radicals that the LDL cholesterol, the triglycerides, and the arterial walls are extensive oxidized. Homocysteine levels are also increased by cigarette smoking which further oxidizes LDL cholesterol and the arterial lining. Oxidation is the initiating cause of atherosclerosis. Therefore, the more and longer one smokes, the more oxidative harm he sustains and the more outstanding his peril of formulating heart disease. The degree of oxidation directly corresponds to the danger of heart disease.

If you are not taking vitamins, minerals, and antioxidants then your LDL cholesterol is being oxidized, it is sticking to your arterial walls, and you ARE formulating heart impairment of normal physiological function EVEN IF YOUR CHOLESTEROL LEVELS ARE NORMAL! LDL cholesterol starts sticking to arterial walls before the age of 5.

Among the a heap of free radicals that harm cholesterol, triglycerides and the arterial lining is homocysteine, a toxic intermediate biochemical devised for the duration of the conversion of the amino acid methionine into another primary amino acid, cysteine. Both methionine and cysteine are non-toxic, but homocysteine is very toxic to the lining of the arterial endothelium. Homocysteine oxidizes LDL cholesterol, triglycerides and the arterial lining.

Homocysteine is an amino acid ordinarily produced in little amounts from the amino acid methionine. The normal role of homocysteine in the body is to control growth and aid bone and tissue formation. However a problem arises when homocysteine levels in the body are elevated, causing exuberant harm to LDL cholesterol, as well as to arteries. Furthermore, homocysteine genuinely stimulates growth of arteriosclerotic plaque, which leads to heart disease.

Thyroid hormone controls the level of homocysteine, but a good deal of constituents play a role in the elevation of homocysteine. Normal aging, kidney failure, smoking, a great deal of medications, and industrial toxins all elevate homocysteine levels. Interestingly, estrogen helps lower homocysteine.

Homocysteine becomes elevated in the blood with a deficiency of the B vitamins-B6, B12 and folic acid. Genetics likewise play a role. About 12% of the population has an undetected defect requiring higher levels of folic acid than the rest of population to help maintain homocysteine levels in a safe range (below 6.5). Therefore if you have high homocysteine levels (> 7.0) even though you are taking supplemental B complex vitamins, then you may be amongst the 12% who need more than 1000 mcg of folic acid per day. In addition, betaine, likewise known as trimethylglycine (TMG) lowers homocysteine.

Homocysteine is second only to cigarette smoking in it is oxidative destruction. It causes little nicks or tears in the arterial lining, while also oxidizing and damaging LDL cholesterol. The damaged, or oxidized LDL cholesterol sticks to the homocysteine-damaged areas of the arterial lining. The combining of oxidized LDL cholesterol and a damaged arterial lining is what causes LDL cholesterol to stick to the arteries, whether or not the LDL cholesterol level is normal.

Cholesterol-lowering statin drugs are the general for treating high cholesterol. This is dogma, and any individual who states other than as supposed or expected is committing medical heresy. Many humans find it hard to believe that pharmaceutical companies could ever succeed in paying medical researchers, medical associations, and doctors to commend something damaging to our health.

Most persons do not recognise that pharmaceutical companies fund medical institutions, medical education, medical conferences, and still reward doctors and exploration foundations for providing favorable results on their drugs. Likewise, pharmaceutical companies often suppress negative results from studies done on their drugs. Money has the power to sweep negative results and severe side effects beneath the rug. Money has the power to influence the FDA to determine which drugs make it to market and which drugs become the “standard” of treatment.

Former editor of the New England Journal of Medicine (NEJM), Dr. Marcia Angell, admonished of the problem of commercializing scientific exploration in her outgoing editorial titled “Is Academic Medicine for Sale?” Angell called for more inviolable limitations on pharmaceutical stock ownership and other financial incentives for researchers. She said that growing conflicts of interest were tainting science, warning “When the boundaries amidst industry and academic medicine become as blurred as they are now, the business goals of industry influence the mission of medical schools in multiple ways.” She did not discount the gains of exploration but said, “a Faustian bargain” now existed amid medical schools and the pharmaceutical industry. Angell left the NEJM in June 2000 and has written a book, “The Truth About the Drug Companies: How They Deceive Us and What to Do About It.”

Two years later, in June 2002, the NEJM declared that it was going to get started accepting articles that were written by biased researchers, as there weren’t sufficient unbiased researchers left to write articles. In other words, most exploration originations were now financed by one or more of the a lot of pharmaceutical companies.

An ABC report cited that a survey of clinical tryouts revealed that when a drug company did not fund a study, favorable results when it comes to a drug were found only 50% of the time. In studies financed by drug companies favorable results in regards to the drugs were reported an awful 90% of the time. Money may and does buy the desired results. This is how most medical exploration and drugs are now invented and brought to market.

In 1977, the internationally-renowned heart surgeon, Dr. Michael DeBakey pointed out that only 30-40% of people with blocked arteries and heart disease have elevated blood cholesterol levels, and posed the logical question, “How do you explain the other 60-70%?”

Because letting down cholesterol did not reduce the peril of death from heart disease, the Cholesterol Consensus Conference in 1984 invented new guidelines to lower the “acceptable level” of cholesterol. High cholesterol would now be the diagnosis for any man or woman with a cholesterol level over 200. Doctors had to convince their persons who requires medical care that they had the impairment of normal physiological function and necessitated to take one or more costly drugs for the rest of their lives.

However, when letting down total cholesterol levels underneath 200 did not translate into saving lives from heart attacks, the focus then turned to LDL cholesterol levels. The “disease” of high cholesterol was refined to the sickness of high LDL cholesterol. The ominous patient who had an LDL cholesterol level above 130 was now condemned to a lifetime of pricey drugs. Though completely illogical, even when a person with normal LDL cholesterol levels suffered a heart attack, he would still be prescribed a cholesterol-lowering drug.

As we shall see, statin drugs reduce the peril of death by repeat heart attacks by as much as 30%, but interestingly enough, the mechanism of action in reducing the risk of death after a heart attack is not thru statin drugs’ capacity to lower cholesterol! It has been came upon that statin drugs have a modest anti-inflammatory and antioxidant effect. Yet, there are a great deal of natural antioxidants that reduce inflammation and oxidation of LDL cholesterol and the lining of the arteries, which may soon be ran into to be more effective in reducing the risk of death than “antioxidant drugs,” without toxic side effects.

The myth that high LDL cholesterol is the indispensable cause of heart disease, and that we must be on drugs to protect ourselves is dispelled by the evidence. If the premise were unfeigned that persons with high levels of LDL cholesterol get heart disease, then we could assume that humans with normal levels of LDL ought to not get heart disease, or at least very few will have to get it. However, as Dr. DeBakey observed, approximately 60% of those who die from heart impairment of normal physiological function have normal LDL cholesterol levels!

Furthermore, after over 45 years of doctors prescribing cholesterol-lowering drugs, heart disease and stroke still stay the number one cause of death in both women and men. This says that disregarding of whether you have a high or a normal level of cholesterol, you have a 50% chance of dying from heart disease. If this is so, and it is, then why take a dangerous drug to try to lower your cholesterol in the basi place?

In 2001, the target level of LDL cholesterol was lowered from 130 to 100, and for the length of one night the number of persons considered to be campaigners for cholesterol statin drugs doubled. Many persons such as myself bristled at the news, because we knew the effectiveness of vitamins, minerals, and antioxidants in preventing and reversing heart disease. Many of us could see the conspiracy for what it was.

The level at which LDL cholesterol is considered normal has continually been influenced by pharmaceutical companies, who pull the financial strings of exploration grants that keep medical schools and medical organizations in business. The lower they may establish the level at which LDL cholesterol is considered to be normal, the more people mechanically become victims of the dreaded impairment of normal physiological function of “high cholesterol.” Therefore, more people will be persuaded that they need to be taking a statin drug, and voilà, more earnings for the manufacturers. When you consider the size of the profits already received, let alone the potential net profit from statin drugs over the next assorted years, the cholesterol conspiracy is one of the biggest cash making systems ever perpetrated on the world.

In July 2004, the level of LDL cholesterol considered normal underwent another change. The new norm plunged from 100 to 70, nearly doubling again the number of humans who are “infected” with the plague of high cholesterol. Why, it’s the epidemic of our time! Many enlightened people howled at this news, marveling if the masses would ever wake up and see who is behind this, and why. Why is the medical institution ignoring the thousands of published medical studies that show the beneficial effects of nutritional supplements versus heart disease? Why is the medical establishment down-playing the dangerous and deadly side effects of statin drugs?

The “updated” LDL cholesterol recommendations were published in the July 2004 issue of the American Heart Association’s publication, Circulation. A panel from the National Heart, Lung and Blood Institute, a division of the National Institutes of Health, which is endorsed by the American College of Cardiology, and the American Heart Association, were the ones who in truth pronounced the new cholesterol level at which drugs must be prescribed. Sounds beauteous official and dependable if these powerful medical foundations are backing up these recommendations, right?

The fact is eight of the nine panel members making the new LDL cholesterol recommendations were being remunerated by the statin-producing pharmaceutical companies. The panelists did not disclose their financial conflict of interest. This info was uncovered by Newsday, a Long Island, New York

newspaper (D. Ricks and R. Robins, Newsday, July 15, 2004). Seven of the nine panelists have financial connections to Pfizer, the makers of Lipitor®. Five of the nine served as “consultants” to Pfizer. So, what did the other two panelists do to is worthy of their money? Seven of the nine panelists also received cash from Merck, the makers of Zocor®, with four of them serving as “consultants” to the company. Eight of the panelists who made the recommendations that would increase the prescribing of statin drugs have received either exploration grants or honoraria from Pfizer, Merck, AstraZeneca, Novartis, Glaxo Smith Kline, Johnson & Johnson, Bayer, and some other drug companies that construct statin drugs.

You would think that with all the promotion and recommendations from medical experts on the gains of statin drugs, the medical community would possess overpowering proof that the drugs reduce the peril of death from cardiovascular disease. A hint of some of the smoke and mirrors in the pharmaceutical companies’ advertising may be seen in their TV commercials. Read conservatively the little print on a heap of of Crestor’s® mercantile advertising. Their mercantile states how much it lowers LDL cholesterol. However, in the same ad you may read, “…Crestor® has not been shown to reduce the risk of heart impairment of normal physiological function or heart attack.” If so, then why take it? Isn’t the bottom line to prevent death?

The scheme for reporting averse effects from medications is tremendously flawed, so much so that a great deal of humans are gravely harmed or killed by a great deal of medications before they are in the long run got rid of from the market. Most doctors do not know what sensations or changes or effects are due to the drug, what will have to be reported, or even to whom to report averse effects. They assume that the exploration that went into developing the drug has already identified all the effects and that a drug brought to market is “safe.” However, only one in twenty side effects is ever reported to either hospital administrators or the FDA.

Statin drugs block cholesterol production in the body by inhibiting the enzyme called HMG-CoA reductase in the early steps of it is synthesis in the mevalonate pathway. Cholesterol is one of three end merchandise in the mevalonate chain. This same biosynthetic pathway is likewise employed to fabricate co-enzyme Q10, or co-Q10, as well as dilochol. Therefore, one ominous consequence of statin drugs is the unintentional inhibition of both Co-Q10 and dilochol synthesis.

The drug data insert of a statin drug states that it lowers co-enzyme Q10 levels. Most doctors have forgotten their biochemistry class in medical school, and forgotten regarding the importance of Co-Q10. Therefore they apparently are not concerned with regards to such a statement on the drug labeling info sheet. They may even reassure their persons who requires medical care that letting down Co-Q10 is not one thing to worry about, but at the same time warn them that the drug may cause liver harm and to have their liver enzymes checked each three to six months to make sure the drug isn’t killing them. They do not realize that it is the depletion of Co-Q10 that leads to liver harm and death.

Ubiquinone, or co-enzyme Q10, is a critical cellular nutrient produced in the cell’s mitochondria, the “engines” that give rise to energy for the cell. Mitochondria use sugar, oxygen, and water to give rise to energy molecules known as ATP. Without ATP cells could do nothing. Damaged tissues could not be repaired. Cells could not divide or manufacture or utilize proteins, enzymes, or hormones. Death of cells, and without doubt of the humane body would take place if ATP could no longer be invented and utilized. Co-Q10 functions within the mitochondria as an electron carrier to cytochrome oxidase, our main respitory enzyme, which helps turn oxygen and sugar into energy. The heart requires high levels of oxygen, sugar, and Co-Q10 since it utilizes a lot of energy. A form of Co-Q10 called ubiquinone is found in all cell membranes, where it plays a role in preserving membrane integrity, so critical to nerve conduction and muscle contraction. Co-Q10 is likewise critical for the formation of elastin and collagen, which make up the connective tissues of the skin, musculature, and the cardiovascular system.

The most mutual side effect of statin drugs is muscle pain and weakness. In fact, a lot of persons who requires medical care who start out on the statin drugs closely without delay detect generalized fatigue and muscle weakness. This is due to the depletion of Co-Q10 necessitated to aid muscle function. Dr. Beatrice Golomb of San Diego, California, is presently conducting a series of studies on statin side effects. The pharmaceutical industry insists that only 2-3% of persons who requires medical care get muscle aches and cramps, when in fact in one study, Golomb found that 98% of people who are in need of medical care taking Lipitor®, and one-third of the persons who requires medical care taking Mevacor® (a lower dose statin), suffered noticeable to significant muscle problems.

Some persons on statin drugs lose coordination of their muscles. Some develop pain in their muscles, some are not capable to write due to loss of fine motor skills. Many lose the strength to exercise. Others are falling more ofttimes as their muscles give out, still others have disturb sleeping due to muscle cramping and twitching. Even worse, some people are experiencing most of these side effects. The difficultnesses are so numerous, it is difficult to list all the sensations or changes humans might experience. These difficulties do not come from the “disease” of high cholesterol, but the sickness of ignorance in prescribing these drugs.

As we age, Co-Q10 levels decline naturally. From the age of 20 to 80, Co-Q10 levels fall by closely 50%. Along with the natural decline of Co-Q10, comes a natural decrease in energy and an increase in the danger of heart disease, stroke, and cancer. If the natural decline of Co-Q10 levels increments the peril of fatigue, cancer, heart disease, and stroke, would it not make sense that accelerating the decline of Co-Q10 levels with statin drugs would have the same effect? They do indeed!

Demonstrating the importance of Co-Q10 to cardiovascular health, in a randomized, double blind, placebo-controlled study of humans either taking or not taking statin drugs, supplementation with Co-Q10 scaled down the risk of heart attacks and death in those with heart sickness and prior heart attacks by 50%, irrespective of whether they were on a statin drug or not. (Singh R, Neki N, Kartikey K, et al. Effect of coenzyme Q10 on danger of atherosclerosis in persons who requires medical care with recent myocardial infarction. Mol Cell Biochem. 2003 Apr; 246(1-2):75-82.)

Additionally, Co-Q10 was shown to increase blood levels of vitamin E and significantly increase the levels of protective HDL. As low HDL is a major danger factor for heart disease, increasing it is a definitive benefit. Statin drugs were shown not to provide any gain beyond that of supplementing with Co-Q10. Let me make this clear – in this study only the co-enzyme Q10 provided any benefit, not the drugs!

Cardiologist Dr. Peter Langsjoen of East Texas University reported the effects of Lipitor® amidst 20 persons who requires medical care who started with wholly normal hearts. After six months on a low dose of 20 mg of Lipitor® per day, two thirds of the people who are in need of medical care started to show signs of heart failure, as seen by abnormalities in the heart’s filling phase. According to Dr. Langsjoen, this malfunction is due to Co-Q10 depletion. Nine controlled tryouts using statin drugs in persons have been conducted thence far. Eight of these showed substantial statin-induced Co-Q10 depletion leading to a decline in left ventricular function and other biochemical imbalances.

In the United States, the incidence of heart attacks over the past ten to fifteen years has declined slightly. But congestive heart failure and cardiomyopathy have risen alarmingly. Is it a coincidence that statin drugs were initial marketed in 1987, and then from 1989 to 1997, deaths from congestive heart failure more than doubled? 38 It frightens me that nearly all people who are in need of medical care with heart failure are put on statin drugs, even if their cholesterol is already low. In my opinion, the worst thing to do for a failing heart is take a statin drug. The best thing is to take is a full range of quality nutritional supplements, …vitamins, minerals, fish oil, and other antioxidants, including Co-Q10.

Various antioxidants work synergistically, each contributing to the fight versus free radicals in dissimilar areas and in dissimilar ways. In the blood stream, water-soluble antioxidants, such as vitamin C, and grape seed extract come in contact with and neutralize free radicals before they harm LDL-cholesterol. Other antioxidants saturate arterial walls and other tissues, and protect collagen and elastic fibers from free radical damage, reducing inflammation and plaque formation. The fat-soluble antioxidants, vitamin E, beta carotene, and co-enzyme Q10 ride along in the blood fat (triglycerides) and LDL cholesterol, protecting them and the endothelium from oxidation. Vitamin E sits on the surface of LDL cholesterol, protecting it from free radical damage. Beta carotene, grape seed extract and olive extract penetrate deeper inside the LDL cholesterol and arterial walls, adding more shelter from oxidation. Quercetin and alpha lipoic acid work through nitrous oxide pathways to reduce high blood pressure, a major peril factor for heart disease.

A report published in the Archives of Internal Medicine in 2005 looked at 97 double-blind controlled studies comparing the efficacy of cholesterol-lowering statin drugs to fish oil. They found that cholesterol-lowering statin drugs scaled down the peril of death from heart disease by only 13%, and

interesting sufficient it was NOT due to the effect of letting down cholesterol. The benefits, even though small, were derived from the fact that statin drugs have a slight antioxidant effect.

Even more interesting, the salmon oil was shown to reduce the peril of death from heart disease by 23%, almost double the gain of statin drugs. Salmon oil is an omega-3 fatty acid that gets integrated into cholesterol and triglycerides and prevents the oxidation of LDL cholesterol. Since LDL cholesterol is protected from exuberant oxidation there is less plaque buildup and less peril of heart disease.

Inflammation is a well-known factor in the formation of atherosclerosis. To keep it simple, think of inflammation and oxidation as the same process. The immune system’s response to inflammation is to

release peroxides that act like acid to break down damaged tissues, so that cells from the immune system, macrophages, may consume the molecules and clean up the site. But peroxides escalate the oxidation/inflammation process, thence damaging more tissue. The arterial walls become more inflamed, escalating the formation of plaque and scarring. The downward cycle proceeds until atherosclerosis is so progressed that the occurrence of a heart attack or stroke becomes imminent.

The liver’s response to inflammation is to release C reactive protein (CRP) into the blood. Other inflammatory causes may cause elevated CRP levels, including cigarette smoking, obesity, insulin insensitivity, diabetes, rheumatoid arthritis, infections, dementia, colorectal cancer, high blood pressure, and aging. Accordingly, elevated CRP levels are a direct indication of inflammation in the body and that atherosclerosis, including heart disease, is actively developing.

Homocysteine and high sensitivity CRP levels may and must be tested. Dr. Jialal, of the Universtity of Texas Southwestern Medical School at Dallas, is well known for his exploration correlating oxidized LDL cholesterol as the true cause of atherosclerosis, has likewise identified high sensitivity C reactive protein as a predictive risk element for inflammation of arterial walls and plaque formation. Your doctor may not test for these routinely, but you ought to insist on getting these tests done. Both of these predictive values may be kept at “safe” levels. Vitamins, minerals, antioxidants, and omega-3 fatty acids may lower the levels of homocysteine and CRP. The B vitamins, along with betaine, or tri-methyl-glycine (TMG), modify homocysteine into safer amino acids and reduce inflammation of the LDL cholesterol and the arterial lining.

When you receive the results of your homocysteine test, do not receive the answer, “Your test was normal.” Ask for the actual number. The doctor and nurse commonly know what is normal by what the lab slip states as the “normal range.” Most lab results report a normal homocysteine level as being beneath 10.4, when in fact, since the early 1990′s, researchers have known that a homocysteine count above 6.5 signals a rapid linear rise in the peril for heart disease.

Furthermore, with each 3 point elevation of homocysteine above 6.5, e.g., when homocysteine levels are 9.5, the danger of coronary artery disease (CAD) rises by an further and added 35%! Yet you may be told that 9.5 is “normal and not to worry.” With a homocysteine level of 12.5, the increase in the

risk for heart sickness surpasses 70%. The dandier the homocysteine level, the dandier the oxidation

of both LDL cholesterol and the arterial lining. The dandier the inflammation, the higher the CRP. Is it any wonder that homocysteine and CRP levels are more predictive for danger of heart impairment of normal physiological function than cholesterol levels and ratios?

I need to emphasize that anybody whether they have a medical problem or not, will have to talk about this selective information with their physician before acting upon anything written here. The selective information provided is not meant to diagnose or treat any disease. It is for informational purposes only; and no one ought to make conclusions in regards to their medications without consulting with their physician. No one must come off a cholesterol-lowering statin drug in lieu of nutritional supplements without a indepth discussion with their physician who is keenly conscious of all the masters and cons of both treatment modalities.

In summary, I commend a full spectrum of quality nutritional supplements, along with a healthful diet and exercise, to support obtain and maintain optimal heart and arterial health. I believe all would agree that modus vivendi changes are the most indispensable factor for optimal health, …and galore believe that quality nutritional supplements are key in protecting versus the procedure that leads to, and accelerates the development of almost all chronic degenerative diseases, that of oxidation. To combat oxidation we need a full range of quality antioxidants.

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The Great Cholesterol Con The Truth About What

Statins are the so-called “wonder drugs” widely prescribed to lower blood cholesterol levels that assert to offer unparalleled shelter versus heart disease. Many experts assert that they are wholly safe and that they are also competent of preventing a whole series of other conditions. This groundbreaking study discloses the truth behind the hype surrounding statins and reveals a number of necessary facts, including that high cholesterol levels do not cause heart disease; that high-fat diets—saturated or otherwise—do not affect blood cholesterol levels; and that for most men and all women the gains offered by statins are negligible at best. Other data is also provided that shows that statins have some more side affects than is oftentimes acknowledged. This hard-hitting survey also points a finger at the powerful pharmaceutical industry and an unquestioning medical profession as perpetrators of the largely facetious conceptions of “good” and “bad” cholesterol that are designed to convince millions of humans to spend billions on statins. With clarity and wit, this appeal to mutual sense and scientific fact debunks mutual assumptions on what constitutes a healthful life style and diet, as well as the idea that there is a miracle heal for heart disease.

Review

“[The Great Cholesterol Con] will save you a lot of heartache—LITERALLY!”  —Examiner.com

Most helpful customer reviews

128 of 129 people found the following review helpful.
Can lowering cholesterol be worse than cholesterol?
By A reader
If you’ve somehow managed to sidestep the pressure to go on statins, this book will provide you with justification. Kendrick walks you, step by step, through your own physiology and bio-chemistry, and backs his contentions that cholesterol can not be the cause of heart disease by citing and summarizing published studies that bear this out. The book is technical but highly readable thanks to an easy conversational style (if your high school biology teacher had been Kendrick, you’d have understood everything and gotten an A). If you don’t really care about arterial plaques and exactly how they’re formed (and exactly how they’re not) the take-away message is pretty much this: statins are ineffective for women, especially for women over 50 years old, and for anybody over 70 years old. Further, statistical studies may indicate that lowering cholesterol encourages cancer. Many of the points Kendrick makes here are also borne out in Gary Taubes’ excellent “Good Calories, Bad Calories.” Both of these books are recommended.

I also feel somewhat compelled to add this: While doctors will tell you they’ve rarely seen anyone with side effects from statins, among my own circle of middle-aged friends, I know 3 who’ve had serious problems with their livers, one who had some muscles permanently destroyed, one–a usually energetic tennis player– who felt, for the few months he took statins, as though he had the flu, and could barely go to work– and one who was left with ringing in the ears and a facial tic. All of these are listed as side effects of statins, as Kendrick points out.

60 of 62 people found the following review helpful.
Another coffin nail for the fat/cholesterol theory of heart disease
By Paul Bergner
It is remarkable that the fat-cholesterol hypothesis of heart disease gained such an established place in US medicine, culture, and popular consciousness, despite a lack of any -strong- evidence to support the theories (including that “bad cholesterol” causes heart disease) and despite sometimes stronger evidence against the theories. The emergence into broader understanding of insulin resistance around the year 2000 was a watershed in the demise of these two theories. I believe the last two months will be looked back on and viewed as the death of these hypotheses.
Perhaps most important, last week results were published that showed that a drug that lowered LDL (“bad”) cholesterol not only did not prevent heart attacks, but may have increased them. The LDL went down, but not the heart attacks. This fairly well disproves the idea that even “bad” cholesterol is really that “bad” in the first place.
There has also been the appearance of two very well researched books on this topic:
Good Calories Bad Calories by Gary Taubes
The Great Cholesterol Con by Malcolm Kendrick (not the same title from Colpo)
Both are impeccable in their science, both show that the fat/cholesterol theory has been, well, frankly, fraudulent from a scientific point of view. Kendrick was lead author of the 14 Countries Study. He took WHO data on fat consumption and heart disease in a large group of countries. From these he selected the seven countries with the lowest fat consumption, and the seven with the highest fat consumption, and compared the rates of heart disease in the two groups. Every one of the countries with the lowest level of fat consumption had a higher rate of heart disease than any of the countries with the highest fat consumption. Do a double take? Read that again.
Taubes goes as far back as 1846 reviewing the science on the cause and cure of obesity (=carbohydrate consumption). He doesn’t miss a stitch.
Both books describe in detail the scientific errors, and false thinking, that led to the acceptance of both hypotheses as if they were Laws, and “settled science” rather than controversial, from s true scientific point of view, from start to finish. Both make good case studies of the methods of good and bad science.
Now we are all going to have to do psychotherapy to treat our obsessive-compulsive fat/cholesterol delusional phobias. But will anyone REALLY stop buying 2% milk instead of whole, or discarding those luscious fatty skin from their chicken breast? I suggest everyone read these two books as part of their psychotherapeutic process.

70 of 75 people found the following review helpful.
excellent
By Paul
Superb science/medical writing. I was already familiar with a lot of the story about cholesterol misinformation, but I still found it very useful to see the issues dissected one-by-one, with comprehensive references to the relevant research studies. The author is obviously extremely well-read in this area, far beyond the main dietary studies. His final chapter about stress and heart-disease is a must-read for anyone interested in these topics, and the fact that he had been so thorough in the earlier part of the book makes me take his speculations seriously. It comes with a good dose of quirky British (actually Scottish) humor, which I enjoyed a lot.

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